Roles of Vitamin A Metabolism in the Development of Hepatic Insulin Resistance

<table class="table-group" width="800px"><tr class="fig-image" id="a"><td><img alt="534972.fig.003a" src="https://static.hindawi.com/articles/isrn/volume-2013/534972/figures/534972.fig.003a.jpg"/></td></tr><tr class="fig-caption"><td align="center"><table><tr><td><b>(a) </b> Insulin-sensitive hepatocytes. </td></tr></table></td></tr><tr class="fig-image" id="b"><td><img alt="534972.fig.003b" src="https://static.hindawi.com/articles/isrn/volume-2013/534972/figures/534972.fig.003b.jpg"/></td></tr><tr class="fig-caption"><td align="center"><table><tr><td><b>(b) </b>Insulin-resistant hepatocytes.</td></tr></table></td></tr></table>

<table>The effects of RA on the insulin-regulated <i>Srebp-1c</i> and <i>Pck1</i> expression levels, the protein levels of SREBP-1c, the expression levels of <i>Raldh1</i> and <i>Fas</i>, and the fatty acid synthesis and gluconeogenesis in insulin-sensitive (a) and insulin-resistant (b) hepatocytes. Please see the text in this paper for description. Up arrows next to the text indicate induction. The intensified weight of the lines indicates the induction of that part of the pathway.</table>

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Figure 3: Roles of Vitamin A Metabolism in the Development of Hepatic Insulin Resistance 